DESCRIPTION: Epigenetic theories of homosexuality concern the studies of changes in gene expression or cellular phenotype caused by mechanisms other than changes in the underlying DNA sequenceand their role in the development of homosexuality. However, epigenetic theories tangle a multiplicity of initiating causes and of resulting final effects and will never lead to a single cause or a single result.Giacomo Zuin: Brazilian Portuguese is way sexyer and smoother
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Photograph by Joel Sartore, National Geographic The biological mechanism for how the metal causes homosexual actions is not totally. Yet in several nations, the laws don't even pretend to protect LGBT . Nunez is a writer and frequent contributor to National Geographic. These celebrations vary in size and scope but share a universal mission of promoting LGBT equality and human rights. They have also become.
Epigenetic theories of homosexuality concern the studies of changes in gene expression or cellular National geographic biology of homosexuality caused by mechanisms other than changes in the underlying DNA sequenceand their role in the development of homosexuality. However, epigenetic theories tangle a multiplicity of initiating causes and of resulting final effects and will never lead to a single cause or a single result. Hence, any interpretation of such theories may not focus just one isolated reason of a multiplicity of National geographic biology of homosexuality or of effects.
DNA in the human body is wrapped around histoneswhich are proteins that package and order DNA into structural units. DNA and histone are covered with chemical tags known as the epigenomewhich shapes the physical structure of the genome. The more tightly wrapped the gene, the less it will be expressed in the organism.
These epigenetic tags react to stimuli presented from the outside world. It adjusts specific genes in the genome to respond to humans' rapidly changing environments. The idea of epigenetics and gene expression has been a theory applied to the origins of homosexuality in humans.
One team of researchers examined the effects of epi-marks buffering XX fetuses and XY fetuses from certain androgen exposure and used published data on fetal androgen signaling and gene regulation through non-genetic changes in DNA packaging to develop a new model for homosexuality.
This theory as well as other concepts involved with epi-marks, twin studiesand fetal androgen signaling will be explored here. Epigenetic marks epi-marks are temporary "switches" that control how our genes are expressed during gestation and after birth. Moreover, epi-marks are modifications of histone proteins. However, can be passed on to offspring through the process of mitosis.
When they are transferred from one parent to an offspring of the opposite sex, it can contribute to an altered sexual development, thus leading to masculinization of female offspring and feminization of male offspring. Identical twins have identical DNA, which leads to the perceived conclusion that all identical twins are either heterosexual or homosexual.
However, it is evident that this is not the case, consequently leaving a gap in the explanation for homosexuality. A "gay" gene does not produce homosexuality. Rather, epigenetic modifications act as temporary "switches" that regulate how the genes are expressed. Epigenetic transformation allows the on and off switching of certain genes, subsequently shaping how cells respond to androgen signaling, which is critical
National geographic biology of homosexuality sexual development.
There are pairs of twins that are discordant with multiple sclerosis and do not both show the trait. After gene testingit was suggested that DNA was identical and that epigenetic differences contributed to the National geographic biology of homosexuality difference between identical twins. While in the fetal stages, hormonal influences of androgenspecifically testosteronecause feminine qualities in regard to sexual development in females and masculine qualities in males.
In typical sexual development, females are exposed to minimal amounts of testosterone, thus feminizing their sexual development, while males are typically exposed to high levels of testosterone, which masculinize their development. Epi-marks play a critical role in this development by acting as a buffer between the fetus and androgen exposure.
Moreover, they predominantly protect XY fetuses from androgen underexposure while protecting XX fetuses from androgen overexposure. The
National geographic biology of homosexuality also suggests that excess androgen exposure in females led to reduced heterosexual interest in adulthood than did females with normal levels of androgen.
New epi-marks are usually produced with each generation, but these marks sometimes carry over between generations. Sex-specific epi-marks are produced National geographic biology of homosexuality early fetal development that protect each sex from the natural disparity in testosterone that occurs during later stages of fetal development.
Different epi-marks protect different sex-specific traits from being masculinized or feminized—some affect the genitals, others affect sexual identity, and yet others affect sexual preference. However, when these epi-marks are transmitted across generations from fathers to daughters or mothers to sons, they may cause reversed effects, such as the feminization of some traits in sons and similarly a partial masculinization of daughters.
Furthermore, the reversed effects of feminization and masculinization can lead to a reversed sexual preference. For example, sex-specific epi-marks normally prevent female fetuses from being masculinized through exposure of atypically high testosteroneand vice versa for male fetuses.
Sex-specific epi-marks are normally erased and not passed between generations. However, they can sometimes escape erasure and are then transferred from a father's genes to a daughter or from a mother's genes to a son. When this happens, this may lead to an altered sexual preference. This demonstrates that gene coding for these epi-marks can spread in the population because they benefit the development and fitness of the parent but only rarely escape erasure, leading to same-sex sexual preference in offspring.
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Rice, Friberg, Urban and Sergey Gavrilets. Homosexuality via canalized sexual development: A testing protocol for a new epigenetic model. Sexually antagonistic epigenetic marks that canalize sexually dimorphic development. The University of Utah, Conway, and Melissa Hines. Retrieved from " https: Views Read Edit View history.
- NatGeo explains how one's sexual orientation is determined during embryonic development. They look at how it is possible for one identical twin to be gay and the other to be straight. The same science explains sexual orientation differences in twins.
- Filmmakers recently went in search of homosexual wild animals as part of a National Geographic Ultimate Explorer documentary about Matthew Grober, biology professor at Georgia State University, agrees, saying, "If [sex].
- While million people live in countries where same-sex relationships are legal, 2.
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